参考文献/References:
[1] Moretti L,Cristofori I,Weaver SM,et al.Cognitive decline in older adults with a history of traumatic brain injury[J].The Lancet Neurology,2012,11(12):1103-1112.
[2] Langlois JA,Rutland-Brown W,Wald MM.The epidemiology and impact of traumatic brain injury:a brief overview[J].J Head Trauma Rehabil,2006,21(5):375-378.
[3] Graham DI,Mcintosh TK,Maxwell WL,et al.Recent advaces in neurotrauma[J].J Neuropathol Exp Neurol,2000,59(8):641-651.
[4] Ai FD.O'leary DM,fan L,Bao W,mullins PG,movsesyan VA[J].Selective blockade of the mGluR1 trauma.Exp Neurol,2001,167(2):435-444.
[5] Huang WD,Fei Z,Zhang X.Traumatic injury induced homer-1a gene expression in cultured cortical neurons of rat[J].Neurosci Lett,2005,389(1):46-50.
[6] Rosonke S,Legome E.Head trauma[J].J emerg Med,2006,31(4):421-425.
[7] 费舟.现代颅脑损伤学[M].北京:人民军医出版社,2007.
[8] Federal Interagency Head Injury Task Force Report.Washington[Z],1989.
[9] Langlois JA,Rutland-Brown W,Tomas K.Traumatic brain injury in the United States:emergency department visits hospitalization and deaths[Z],2004.
[10] Thurman DJ,Alverson C,Dunn KA,et al.Traumatic brain injury in the United States:A public health perspective[J].J Head Trauma Rehabil,1999,14(6):602-615.
[11] Zhao YD,Wang W.Neurosurgical trauma in People's Republic of China[J].World J Surg,2001,25(9):1202-1204.
[12] Rink A,Fung KM,Trojanowski JQ,et al.Evidence of apoptosis cell death after experience traumatic brain injury in rat[J].Am J Pathol,1995,147(6):1575.
[13] Conti AC,Raghupathi R,Trojanowski JQ,et al.Experimental brain injury induces regionally distinct apoptosis during the acute and delayed post-traumatic period[J].J Neurosci,1998,18(15):5663-5672.
[14] Williams S,Raghupathi R,Mackinnon MA,et al.In situ DNA fragmentation occurs in white matter up to 12 months after head injury in man[J].Acta Neuropathol,2001,102(6):581-590.
[15] 雪亮,杨树源.人脑创伤后神经元凋亡及调节机制的观察[J].中华创伤杂志,2003,19(3):160-163.
[16] Degterev A,Huang ZH,Boyce M,et al.Chemical inhibitor of nonapoptotic cell death with therapeutic potential for ischemic brain injury[J].Nat Chem Biol,2005,1(2):112-119.
[17] Han WD,Xie JS,Li L,et al.Necrostatin-1 reverts shikonin-induced necroptosis to apoptosis[J].Apoptosis,2009,14(5):674-686.
[18] Xu X,Chua CC,Kong J,et al.Necrostatin-1 protects against glutamate-induced glutathione depletion and caspase-independent cell death in HT-22 cells[J].J Neurochem,2007,103(5):2004-2014.
[19] Xu X,Chua KW,Chua CC,et al.Synergistic protective effects of humanin and necrostatin-1 on hypoxia and ischemia/reperfusion injury[J].Brain Res,2010,1355(2):189-194.
[20] Rosenbaum DM,Degterev A,David J,et al.Necroptosis,a novel form of caspase-independent cell death,contributes to neuronal damage in a retinal ischemia-reperfusion injury model[J].J Neurosci Res,2010,88(7):1569-1576.
[21] Lin MT,Beal MF.Mitochondrial dysfunction and oxidative stress in neurodegenerative diseases[J].Nature,2006,443(7113):787-795.
[22] Li Y,Yang X,Ma C,et al.Necroptosis contributes to the NMDA-induced excitotoxicity in rat's cultured cortical neurons[J].Neurosci Lett,2008,447(2/3):120-123.
[23] Zhu S,Zhang Y,Bai G,et al.Necrostatin-1 ameliorates symptoms in R6/2 transgenic mouse model of Huntington's disease[J].Cell Death Dis,2011,2(1):e115.
[24] Pascual MJ,Reeds PJ.In vivo glucose contribution to glutamate synthesis is maintained while its contribution to acetyl CoA is lowered in adult mice fed a restricted amount of carbohydrate J[J].Nutr,1998,128(128):733-739.
[25] Liu J,Wu DC,Wang YT.Allosteric potentiation of glycine receptor chloride currents by glutamate[J].Nat Neurosci,2010,13(10):1225-1232.