[1]吴迪 许凤燕 孙亮 唐旭东 JPP Kumal 毛东升 李怡萱 吴树亮 刘彬.外伤性脑损伤诱发阿尔茨海默病的相关机制研究进展[J].卒中与神经疾病杂志,2019,26(03):381-384.[doi:10.3969/j.issn.1007-0478.2019.03.034]
点击复制

外伤性脑损伤诱发阿尔茨海默病的相关机制研究进展()
分享到:

《卒中与神经疾病》杂志[ISSN:1007-0478/CN:42-1402/R]

卷:
第26卷
期数:
2019年03期
页码:
381-384
栏目:
综 述
出版日期:
2019-06-25

文章信息/Info

文章编号:
1007-0478(2019)03-0381-04
作者:
吴迪 许凤燕 孙亮 唐旭东 JPP Kumal 毛东升 李怡萱 吴树亮 刘彬
150001 哈尔滨医科大学附属第四医院神经内科[吴迪 刘彬(通信作者)]; 哈尔滨医科大学人体解剖学教研室[许凤燕 孙亮 唐旭东 JPP Kumal 毛东升 李怡萱 吴树亮(通信作者)]
分类号:
R742
DOI:
10.3969/j.issn.1007-0478.2019.03.034
文献标志码:
A

参考文献/References:

[1] Dewan MC, Rattani A, Gupta S, et al. Estimating the global incidence of traumatic brain injury[J]. J Neurosurg, 2018,1-18.
[2] Hawley L, Hart T, Waldman W, et al. Living well after traumatic brain injury[J]. Archives of Physical Medicine and Rehabilitation, 2018, 99(7):1441-1442.
[3] Timmons SD. The challenges of estimating the cost of traumatic brain injury worldwide[J]. Neurosurgical Focus, 2018, 44(5):1-2.
[4] Alzheimer's Association. 2016 Alzheimer's disease facts and figures[J]. Alzheimers Dement, 2016,12(4):459-509.
[5] Readhead B, Haure-Mirande JV, FUNK CC, et al. Multiscale analysis of Independent alzheimer's cohorts finds disruption of molecular,genetic,and clinical networks by human herpesvirus[J]. Neuron, 2018, 99(1):64-82.
[6] Da Mesquita-s, Louveau A,Vaccari A,et al.Functional aspects of meningeal lymphatics in ageing and Alzheimer's disease[J]. Nature, 2018, 560(7717):185-191.
[7] Abbasi J. Promising results in 18-Month analysis of alzheimer drug candidate[J]. JAMA-Journal of the American Medical Association, 2018, 320(10):965.
[8] Mortimer JA, Vanduijn CM, Chandra V, et al. Head trauma as a risk factor for alzheimers-disease - a collaborative reanalysis of case-control studies[J]. International Journal of Epidemiology, 1991, 20(2):S28-S35.
[9] Shishido H, Kishimoto Y, Kawai N, et al. Traumatic brain injury accelerates amyloid-beta deposition and impairs spatial learning in the triple-transgenic mouse model of Alzheimer's disease[J]. Neuroscience Letters, 2016, 629:62-67.
[10] Weiner MW, Crane PK, Montine TJ, et al. Traumatic brain injury May not increase the risk of Alzheimer disease[J]. Neurology, 2017, 89(18):1923-1925.
[11] Julien J, Joubert S, Ferland MC, et al. Association of traumatic brain injury and Alzheimer disease onset: A systematic review[J]. Annals of Physical and Rehabilitation Medicine, 2017, 60(5, SI):347-356.
[12] Crane PK, Gibbons LE, Dams-O'connor KA, et al. Association of traumatic brain injury with Late-Life neurodegenerative conditions and neuropathologic findings[J]. JAMA Neurology, 2016, 73(9):1062-1069.
[13] Parker HL, Traumatic Encephalopathy.(`Punch drunk')of professional pugilists[J]. The Journal of Neurology and Psychopathology, 1934, 15(57):20-28.
[14] Rudelli R, Strom JO, Welch PT, et al. Posttraumatic premature Alzheimer's disease.Neuropathologic findings and pathogenetic considerations[J]. Archives of Neurology, 1982, 39(9):570-575.
[15] Hayes J, Logue M, Sadeh N, et al. Mild traumatic brain injury is associated with reduced cortical thickness in those at risk for Alzheimer's disease[J]. Brain Injury, 2017, 31(6/7):885-886.
[16] Plassman BL, Havlik RJ, Steffens DC, et al. Documented head injury in early adulthood and risk of Alzheimer's disease and other dementias[J]. Neurology, 2000, 55(8):1158-1166.
[17] Nemetz PN, Leibson C, Naessens JM, et al. Traumatic brain injury and time to onset of Alzheimer's disease:a population-based study[J]. American Journal of Epidemiology, 1999, 149(1):32-40.
[18] Lobue C, Wadsworth H, Wilmoth K, et al. Traumatic brain injury history is associated with earlier age of onset of Alzheimer disease[J]. Clinical Neuropsychologist, 2017, 31(1):85-98.
[19] Yang Shun-tai, Hsiao I T, Hsieh C J, et al. Accumulation of amyloid in cognitive impairment after mild traumatic brain injury[J]. Journal of the Neurological Sciences, 2015, 349(1/2):99-104.
[20] Hu W, Yc T, Zhang Y, et al. Involvement of activation of asparaginyl endopeptidase in TauHyperphosphorylation in repetitive mild traumatic brain injury[J]. Journal of Alzheimer's Disease: JAD, 2018, 64(3):709-722.
[21] 吴琼,温咪咪,李娜,等.淀粉样前体蛋白生理功能研究进展[J].生理科学进展,2016,47(2):103-107.
[22] Van DEN Heuvel C, Blumbergs P C, FINNIE J W, et al. Upregulation of amyloid precursor protein messenger RNA in response to traumatic brain injury:an ovine head impact model[J]. Experimental Neurology, 1999, 159(2):441-450.
[23] Blasko I, Beer R, Bigl M, et al. Experimental traumatic brain injury in rats stimulates the expression,production and activity of Alzheimer's disease beta-secretase(BACE-1)[J]. J Neural Transm(Vienna), 2004, 111(4):523-536.
[24] Washington PM, Morffy N, Parsadanian M, et al. Experimental traumatic brain injury induces rapid aggregation and oligomerization of amyloid-beta in an Alzheimer's disease mouse model[J]. Journal of Neurotrauma, 2014, 31(1):125-134.
[25] Abisambra JF, Scheff S. Brain injury in the context of tauopathies[J]. Journal of Alzheimer's Disease:JAD, 2014, 40(3):495-518.
[26] Liu CC, Liu CC, Kanekiyo T, et al. Apolipoprotein E and alzheimer disease: risk, mechanisms and therapy[J]. Nature Reviews Neurology, 2013, 9(2):106-118.
[27] Merritt VC, Clark AL, Sorg SF, et al. Apolipoprotein E(APOE)ε4 genotype is associated with reducedneuropsychological performance in military veterans with a history of mildtraumatic brain injury[J]. Journal of Clinical and Experimental Neuropsychology, 2018, 40(10):1050-1061.
[28] Washington PM, Burns MP. The effect of the APOE4 gene on accumulation of Aβ40 after brain Injury Cannot be reversed by increasing apoE4 protein[J]. Journal of Neuropathology and Experimental Neurology, 2016, 75(8):770-778.
[29] Hirsch-Reinshagen V, Burgess BL, Wellington CL. Why lipids are important for Alzheimer disease?[J]. Molecular and Cellular Biochemistry, 2009, 326(1/2):121-129.
[30] Fitz NF, Cronican AA, Saleem M, et al. Abca1 deficiency affects Alzheimer's disease-like phenotype in human ApoE4 but not in ApoE3-targeted replacement mice[J]. Journal of Neuroscience, 2012, 32(38):13125-13136.
[31] Castranio EL, Wolfe CM, Nam KN, et al. ABCA1 haplodeficiency affects the brain transcriptome following traumatic brain injury in mice expressing human APOE isoforms[J]. Acta Neuropathologica Communications, 2018, 6(1):69.
[32] Shi Y, Yamada K, Liddelow SA, et al. ApoE4 markedly exacerbates tau-mediated neurodegeneration in a mouse model of tauopathy[J]. Nature, 2017, 549(7673):523.
[33] Bennett RE, Esparza TJ, Lewis HA, et al. Human apolipoprotein E4 worsens acute axonal pathology but not amyloid-beta immunoreactivity after traumatic brain injury in 3xTG-AD mice[J]. Journal of Neuropathology and Experimental Neurology, 2013, 72(5):396-403.
[34] Giunta B, Obregon D, Velisetty R, et al. The immunology of traumatic brain injury: a prime target for Alzheimer'sdisease prevention[J]. J Neuroinflammation, 2012, 9:185.
[35] Jiang T, Tan L, Zhu XC, et al. Upregulation of TREM2 ameliorates neuropathology and rescues spatialcognitive impairment in a transgenic mouse model of Alzheimer's disease[J]. Neuropsychopharmacology, 2014, 39(13):2949-2962.
[36] Wang Y, Cella M, Mallinson K, et al. TREM2 lipid sensing sustains the microglial response in an Alzheimer's diseasemodel[J]. Cell, 2015, 160(6):1061-1071.
[37] Saber M, Kokiko-Cochran O, Puntambekar S S, et al. Triggering receptor expressed on myeloid cells 2 deficiency alters acute macrophage distribution and improves recovery after traumatic brain injury[J]. Journal of Neurotrauma, 2017, 34(2):423-435.
[38] Acosta C, Anderson HD, Anderson CM. Astrocyte dysfunction in alzheimer disease[J]. Journal of Neuroscience Research, 2017, 95(12):2430-2447.
[39] Nedergaard M. Garbage truck of the brain[J]. Science, 2013, 340(6140):1529-1530.
[40] Iliff JJ, Nedergaard M. Is there a cerebral lymphatic system?[J]. Stroke, 2013, 44(6, 1):S93-S95.
[41] Iliff JJ, Chen MJ, Plog B A, et al. Impairment of glymphatic pathway function promotes Tau pathology after traumatic brain injury[J]. Journal of Neuroscience, 2014, 34(49):16180-16193.
[42] Cheng WH, Stukas S, Martens KM, et al. Age at injury and genotype modify acute inflammatory and neurofilament-light responses to mild CHIMERA traumatic brain injury in wild-type and APP/PS1mice[J]. Exp Neurol, 2018, 301(Pt A):26-38.
[43] D'anna L, Abu-Rumeileh S, Fabris M, et al. Serum interleukin-10 levels correlate with cerebrospinal fluid amyloid BetaDeposition in Alzheimer disease patients[J]. Neuro-degenerative Diseases, 2017, 17(4/5):227-234.
[44] Shamim D, Laskowski M. Inhibition of inflammation mediated through the tumor necrosis factor α biochemical pathway can lead to favorable outcomes in Alzheimer disease[J]. J Cent Nerv Syst Dis,2017, 9:1-10.
[45] Ramos-Cejudo J, Wisniewski T, Marmar C A, et al. Traumatic brain injury and alzheimer's disease: the cerebrovascular Link[J]. EBIOMEDICINE, 2018, 28:21-30.

更新日期/Last Update: 2019-06-25