[1]杜仁峰 陈阳 林富春 周杭 杨化兰 杨丹 李清华.免疫细胞与缺血性脑卒中的相关性研究进展[J].卒中与神经疾病杂志,2021,28(03):346-350.[doi:10.3969/j.issn.1007-0478.2021.03.020]
点击复制

免疫细胞与缺血性脑卒中的相关性研究进展()
分享到:

《卒中与神经疾病》杂志[ISSN:1007-0478/CN:42-1402/R]

卷:
第28卷
期数:
2021年03期
页码:
346-350
栏目:
综述
出版日期:
2021-06-25

文章信息/Info

文章编号:
1007-0478(2021)03-0346-05
作者:
杜仁峰 陈阳 林富春 周杭 杨化兰 杨丹 李清华
作者单位:201399 上海市浦东医院(复旦大学附属浦东医院)[杜仁峰 陈阳 林富春 周杭 杨化兰 杨丹 李清华(通信作者)]
分类号:
R743.3
DOI:
10.3969/j.issn.1007-0478.2021.03.020
文献标志码:
A

参考文献/References:

[1] Li Z, Bai Y, Li WB, et al. Carotid vulnerable plaques are associated with circulating leukocytes in acute ischemic stroke patients: an clinical study based on contrast-enhanced ultrasound[J]. Sci Rep, 2018, 8(1): 8849.
[2] Jian Z, Liu R, Zhu XQ, et al. The involvement and therapy target of immune cells after ischemic stroke[J]. Front Immunol, 2019, 10: 2167.
[3] Ao LY, Yan YY, Zhou L, et al. Immune cells after ischemic stroke onset: roles, migration, and target intervention[J]. J Mol Neurosci, 2018, 66(3): 342-355.
[4] Rajkovic O, Potjewyd G, Pinteaux E. Regenerative medicine therapies for targeting neuroinflammation after stroke[J]. Front Neurol, 2018, 9: 734.
[5] Yang C, Hawkins KE, Doré S, et al. Neuroinflammatory mechanisms of blood-brain barrier damage in ischemic stroke[J]. Am J Physiol Cell Physiol, 2019, 316(2): C135-C153.
[6] Zhang SX. Microglial activation after ischaemic stroke[J]. Stroke Vasc Neurol, 2019, 4(2): 71-74.
[7] Guruswamy R, Elali A. Complex roles of microglial cells in ischemic stroke pathobiology: new insights and future directions[J]. Int J Mol Sci, 2017, 18(3): 496.
[8] Qin C, Zhou LQ, Ma XT, et al. Dual functions of microglia in ischemic stroke[J]. Neurosci Bull, 2019, 35(5): 921-933.
[9] Thurgur H, Pinteaux E. Microglia in the neurovascular unit: Blood-Brain barrier-microglia interactions after central nervous system disorders[J]. Neuroscience, 2019, 405(6): 55-67.
[10] Camargos QM, Silva BC, Silva DG, et al. Minocycline treatment prevents depression and anxiety-like behaviors and promotes neuroprotection after experimental ischemic stroke[J]. Brain Res Bull, 2020, 155(11): 1-10.
[11] Zhao SC, Ma LS, Chu ZH, et al. Regulation of microglial activation in stroke[J]. Acta Pharmacol Sin, 2017, 38(4): 445-458.
[12] Sandvig I, Augestad IL, H?erg AK, et al. Neuroplasticity in stroke recovery. The role of microglia in engaging and modifying synapses and networks[J]. Eur J Neurosci, 2018, 47(12): 1414-1428.
[13] Pekny M, Wilhelmsson U, Tatlisumak T, et al. Astrocyte activation and reactive gliosis-A new target in stroke?[J]. Neurosci Lett, 2019, 689(7): 45-55.
[14] Jayaraj RL, Azimullah S, Beiram R, et al. Neuroinflammation: friend and foe for ischemic stroke[J]. J Neuroinflammation, 2019, 16(1): 142.
[15] Liu Z, Chopp M. Astrocytes, therapeutic targets for neuroprotection and neurorestoration in ischemic stroke[J]. Prog Neurobiol, 2016, 144(9): 103-120.
[16] Siracusa R, Fusco R, Cuzzocrea S. Astrocytes: role and functions in brain pathologies[J]. Front Pharmacol, 2019, 10: 1114.
[17] Wang X, Xuan W, Zhu ZY, et al. The evolving role of neuro-immune interaction in brain repair after cerebral ischemic stroke[J]. CNS Neurosci Ther, 2018, 24(12): 1100-1114.
[18] Weisenburger-Lile D, Dong Y, Yger M, et al. Harmful neutrophil subsets in patients with ischemic stroke: Association with disease severity[J]. Neurol Neuroimmunol Neuroinflamm, 2019, 6(4): e571.
[19] Ruhnau J, Schulze J, Dressel A, et al. Thrombosis, neuroinflammation, and poststroke infection: the multifaceted role of neutrophils in stroke[J]. J Immunol Res, 2017: 5140679.
[20] Shi Y, Zhang LL, Pu HJ, et al. Rapid endothelial cytoskeletal reorganization enables early blood-brain barrier disruption and long-term ischaemic reperfusion brain injury[J]. Nat Commun, 2016, 7: 10523.
[21] Turner RJ, Sharp FR. Implications of MMP9 for blood brain barrier disruption and hemorrhagic transformation following ischemic stroke[J]. Front Cell Neurosci, 2016, 10: 56.
[22] Filiano AJ, Gadani SP, Kipnis J. How and why do T cells and their derived cytokines affect the injured and healthy brain?[J]. Nat Rev Neurosci, 2017, 18(6): 375-384.
[23] Mracsko E, Liesz A, Stojanovic A, et al. Antigen dependently activated cluster of differentiation 8-positive T cells cause perforin-mediated neurotoxicity in experimental stroke[J]. J Neurosci, 2014, 34(50): 16784-16795.
[24] Ito M, Komai K, Nakamura T, et al. Tissue regulatory T cells and neural repair[J]. Int Immunol, 2019, 31(6): 361-369.
[25] Wang Y, Zhang JH, Sheng JF, et al. Immunoreactive cells after cerebral ischemia[J]. Front Immunol, 2019, 10: 2781.
[26] Noh MY, Wm L, Lee SJ, et al. Regulatory T cells increase after treatment with poly(ADP-ribose)polymerase-1 inhibitor in ischemic stroke patients[J]. Int Immunopharmacol, 2018, 60(4): 104-110.
[27] Selvaraj UM, Poinsatte K, Torres V, et al. Heterogeneity of B cell functions in Stroke-Related risk, prevention, injury, and repair[J]. Neurotherapeutics, 2016, 13(4): 729-747.
[28] Doyle KP, Quach LN, Solé M, et al. B-lymphocyte-mediated delayed cognitive impairment following stroke[J]. J Neurosci, 2015, 35(5): 2133-2145.
[29] Prüss H, Iggena D, Baldinger T, et al. Evidence of intrathecal immunoglobulin synthesis in stroke: a cohort study[J]. Arch Neurol, 2012, 69(6): 714-717.
[30] Kalev-Zylinska ML, Symes W, Little KC, et al. Stroke patients develop antibodies that react with components of N-methyl-D-aspartate receptor subunit 1 in proportion to lesion size[J]. Stroke, 2013, 44(8): 2212-2219.
[31] Chen C, Ai QD, Chu SF, et al. NK cells in cerebral ischemia[J]. Biomedicine & Pharmacotherapy, 2019, 109(10): 547-554.
[32] Spahn JH, Li W, Kreisel D. Innate immune cells in transplantation[J]. Curr Opin Organ Transplant, 2014, 19(1): 14-19.
[33] Rayasam A, Hsu M, Kijak JA, et al. Immune responses in stroke: how the immune system contributes to damage and healing after stroke and how this knowledge could be translated to better cures?[J]. Immunology, 2018, 154(3): 363-376.
[34] Zhou W, Liesz A, Bauer H, et al. Postischemic brain infiltration of leukocyte subpopulations differs among murine permanent and transient focal cerebral ischemia models[J]. Brain Pathol, 2013, 23(1): 34-44.
[35] Jones KA, Maltby S, Plank MW, et al. Peripheral immune cells infiltrate into sites of secondary neurodegeneration after ischemic stroke[J]. Brain Behav Immun, 2018, 67(9): 299-307.
[36] Chu HX, Kim HA, Lee S, et al. Immune cell infiltration in malignant middle cerebral artery infarction: comparison with transient cerebral ischemia[J]. J Cereb Blood Flow Metab, 2014, 34(3): 450-459.
[37] Mao L, Li PY, Zhu W, et al. Regulatory T cells ameliorate tissue plasminogen activator-induced brain haemorrhage after stroke[J]. Brain, 2017, 140(7): 1914-1931.
[38] Ito M, Komai K, Mise-Omata S, et al. Brain regulatory T cells suppress astrogliosis and potentiate neurological recovery[J]. Nature, 2019, 565(7738): 246-250.
[39] Kristian PD, Buckwalter MS. Does B lymphocyte-mediated autoimmunity contribute to post-stroke dementia?[J]. Brain Behav Immun, 2017, 64(8): 1-8.
[40] Gan Y, Liu Q, Wu W, et al. Ischemic neurons recruit natural killer cells that accelerate brain infarction[J]. Proc Natl Acad Sci U S A, 2014, 111(7): 2704-2709.
[41] Zhang Y, Gao Z, Wang D, et al. Accumulation of natural killer cells in ischemic brain tissues and the chemotactic effect of IP-10[J]. J Neuroinflammation, 2014, 11(1): 79.
[42] Ludewig P, Gallizioli M, Urra X, et al. Dendritic cells in brain diseases[J]. Biochim Biophys Acta, 2016, 1862(3): 352-367.
[43] Chen C, Chencheng Z, Cuiying L, et al. Plasmacytoid dendritic cells protect against middle cerebral artery occlusion induced brain injury by priming regulatory T cells[J]. Front Cell Neurosci, 2020, 14: 8.
[44] Petrovic-Djergovic D, Goonewardena SN, Pinsky DJ. Inflammatory disequilibrium in stroke[J]. Circ Res, 2016, 119(1): 142-158.

备注/Memo

备注/Memo:
基金项目:上海市浦东新区卫生系统重点学科群建设资助(项目编号为PWZ-xq2017-09)
更新日期/Last Update: 2021-06-25