参考文献/References:
[1] Worley OR,Baugh MD,Hughes DA,et al.Metalloproteinase expression in PMA-stimulated THP-1 cells. Effects of peroxisome proliferator-activated receptor-γ(PPAR-γ)agonists and 9-cis-retinoic acid[J].J Biol Chem,2003,278(51):51340-51346.
[2] 王利,王宪,孔炜.新型金属蛋白酶ADAMTS家族的研究进展[J].生理科学进展,2008,39(1):49-52.
[3] Wågsäter D,Björk H,Zhu C,et al.ADAMTS-4 and -8 are inflammatory regulated enzymes expressed in macrophage-rich areas of human atherosclerotic plaques[J].Atherosclerosis,2008,196(2):514-522.
[4] Salter RC,Ashlin TG,Kwan AP,et al.ADAMTS proteases: key roles in atherosclerosis?[J].J Mol Med(Berl),2010,88(12):1203-1211.
[5] 王勇.ADAMTS-1在老年大鼠心肌缺血预适应中的作用[D].武汉:武汉大学,2011.
[6] Jonnsson-Rylander AC,Nilsson T,Fritsche-Danielson RA,et al.Role of ADAMTS-1 in atherosclerosis - Remodeling of carotid artery, immunohistochemistry, and proteolysis of versican[J].Arterioscler Thromb Vasc Biol,2005,25(1):180-185.
[7] Lyu C,Chen Y,Zhu M,et al. Association of ADAMTS-1 gene polymorphisms with ischemic stroke caused by large artery atherosclerosis [J].Zhonghua Yi Xue Yi Chuan Xue Za Zhi,2015,32(6):844-848.
[8] Liu CJ,Kong W,Ilalov K,et al.ADAMTS-7: a metalloproteinase that directly binds to and degrades cartilage oligomeric matrix protein[J].FASEB J,2006,20(7):988-990.
[9] Riessen R,Fenchel M,Chen H,et al.Cartilage oligomeric matrix protein(thrombospondin-5)is expressed by human vascular smooth muscle cells[J].Arterioscler Thromb Vasc Biol,2001,21(1):47-54.
[10] Wang L,Zheng J,Bai X,et al.ADAMTS-7 mediates vascular smooth muscle cell migration and neointima formation in balloon-injured rat arteries[J].Circ Res,2009,104(5):688-698.
[11] Zhang L,Yu F,Wang L,et al.ADAMTS-7 promotes vascular smooth muscle cells proliferation in vitro and in vivo[J].Sci China Life Sci,2015,58(7):674-681.
[12] Reilly MP,Li M,He J,et al.Identification of ADAMTS7 as a novel locus for coronary atherosclerosis and association of ABO with myocardial infarction in the presence of coronary atherosclerosis: two genome-wide association studies[J].Lancet,2011,377(9763):383-392.
[13] Van Setten J,Isgum I,Smolonska J,et al.Genome-wide association study of coronary and aortic calcification implicates risk loci for coronary artery disease and myocardial infarction[J].Atherosclerosis,2013,228(2):400-405.
[14] Newby AC.Proteinases and plaque rupture: unblocking the road to translation[J].Curr Opin Lipidol,2014,25(5):358-366.
[15] You L,Tan L,Liu L,et al.ADAMTS7 locus confers high cross-race risk for development of coronary atheromatous plaque[J].Mol Genet Genomics,2016,291(1):121-128.
[16] Bayoglu B,Arslan C,Tel C,et al.Genetic variants rs1994016 and rs3825807 in ADAMTS7 affect its mRNA expression in atherosclerotic occlusive peripheral arterial disease[J].J Clin Lab Anal,2017,31(5):366-369.
[17] 刘鹏,何欣威,朱敏,等.含Ⅰ型血小板反应蛋白的解聚素和金属蛋白酶-12基因多态性与大动脉粥样硬化型脑梗死的关系[J].中华神经科杂志,2015,48(10):855-860.
[18] Furlan M,Robles R,Galbusera M,et al.Von willebrand factor-cleaving protease in thrombotic thrombocytopenic purpura and the hemolytic-uremic syndrome[J].N Engl J Med,1998,339(22):1578-1584.
[19] Tsai HM,Lian EC.Antibodies to von willebrand factor-cleaving protease in acute thrombotic thrombocytopenic purpura[J].N Engl J Med,1998,339(22):1585-1594.
[20] Sonneveld MA,De Maat MP,Portegies ML,et al.Low ADAMTS13 activity is associated with an increased risk of ischemic stroke[J].Blood,2015,126(25):2739-2746.
[21] Akyol O,Akyol S,Chen CH.Update on ADAMTS13 and VWF in cardiovascular and hematological disorders[J].Clin Chim Acta,2016,463(12):109-118.
[22] Sonneveld MA,Franco OH,Ikram MA,et al.Von willebrand factor, ADAMTS13, and the risk of mortality: the Rotterdam study[J].Arterioscler Thromb Vasc Biol,2016,36(12):2446-2451.
[23] Zhao L,Hua C,Li Y,et al.miR-525-5p inhibits ADAMTS13 and is correlated with ischemia/reperfusion injury-induced neuronal cell death[J].Int J Clin Exp Med,2015,8(10):18115-18122.
[24] Xu H,Cao Y,Yang X,et al.ADAMTS13 controls vascular remodeling by modifying VWF reactivity during stroke recovery[J].Blood,2017,130(1):11-22.
[25] Apte SS.A disintegrin-like and metalloprotease(reprolysin type)with thrombospondin type 1 motifs: the ADAMTS family[J].Int J Biochem Cell Biol,2004,36(6):981-985.
[26] Cross AK,Haddock G,Stock CJ,et al.ADAMTS-1 and -4 are up-regulated following transient middle cerebral artery occlusion in the rat and their expression is modulated by TNF in cultured astrocytes[J].Brain Res,2006,1088(1):19-30.
[27] Bradbury EJ,Carter LM.Manipulating the glial scar: chondroitinase ABC as a therapy for spinal cord injury[J].Brain Res Bull,2011,84(4/5):306-316.
[28] Hill JJ,Jin KL,Mao XO,et al.Intracerebral chondroitinase ABC and heparan sulfate proteoglycan glypican improve outcome from chronic stroke in rats[J].Proc Natl Acad Sci U S A,2012,109(23):9155-9160.
[29] Soleman S,Yip PK,Duricki DA,et al.Delayed treatment with chondroitinase ABC promotes sensorimotor recovery and plasticity after stroke in aged rats[J].Brain,2012,135(Pt 4):1210-1223.
[14].Krstic D, Rodriguez M, Knuesel I. Regulated Proteolytic Processing of Reelin through Interplay of Tissue Plasminogen Activator(tPA), ADAMTS-4, ADAMTS-5, and Their Modulators[J]. Plos One, 2012, 7(7):e47793.
[30] Krstic D,Rodriguez M,Knuesel I.Regulated proteolytic processing of Reelin through interplay of tissue plasminogen activator(tPA), ADAMTS-4, ADAMTS-5, and their modulators[J].PLoS One,2012,7(10):e47793.
[31] Zamanian JL,Xu L,Foo LC,et al.Genomic analysis of reactive astrogliosis[J].J Neurosci,2012,32(18):6391-6410.
[16].Lemarchant S, Pomeshchik Y, Kidin I, et al. ADAMTS-4 promotes neurodegeneration in a mouse model of amyotrophic lateral sclerosis[J]. Molecular Neurodegeneration, 2016, 11(1):10.
[32] Lemarchant S,Pomeshchik Y,Kidin I,et al.ADAMTS-4 promotes neurodegeneration in a mouse model of amyotrophic lateral sclerosis[J].Mol Neurodegener,2016,11(1):10.
[33] Lemarchant S,Wojciechowski S,Vivien D,et al.ADAMTS-4 in central nervous system pathologies[J].J Neurosci Res,2017,95(9):1703-1711.